Air embolism – central line disruption

Theory

Subclinical air embolism is common. Symptomatic air embolism post CVC line insertion <2%.

Small amount air – subclinical.

Intermediate amounts:

Large amount (3–8 ml/kg) – acute right ventricular outflow obstruction, cardiogenic shock and circulatory collapse.

In setting of ASD/VSD/patent foramen ovale, even small amounts of air can cause peripheral embolisation to territory supplied by blood vessel.

Immediate intern management

Attend patient and make rapid assessment.

If patient unconscious/severe respiratory distress/arrest

  1. Tell nursing staff to call Code Blue then move to head end of bed.
  2. Triple manoeuvre – chin lift, jaw thrust, head tilt.
  3. Bag and mask patient with oxygen.
  4. Start CPR if arrested.

Patient conscious/mild distress

  1. Stop central line infusion/clamp line.
  2. Give 100% oxygen by mask and place patient in trendelenburg position (head down).
  3. If patient increasingly distressed:
    • Rotate to left hand side with right side facing upwards
      (left lateral position). (Traps air at apex of ventricle).
    • Ask nursing staff to call MET code.
  1. Get crash trolley, obtain IV access and send off blood tests.
  2. Notify unit registrar, ICU registrar and anaesthetist on call.

Clinical features (obtain rapidly)

History

Examination

Other (obtain later)

Investigations

  1. ABG
  1. ECG
  1. CXR
  1. Basic blood tests

Further definitive management

Immediate cardiorespiratory support and resuscitation

Remember there are more common causes for acute SOB in a patient with a central line: for example, pneumothorax, pulmonary embolus, acute pulmonary oedema, sputum retention and anaphylaxis. Initial assessment should be aimed at ruling out these other causes and then, if they are not present, considering the possibility of air embolus.

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